Physiology
II - PB 3325
Course
Director: Mrs. Kay Brashear
STUDY GUIDE QUIZ 2
LAB MANUAL: EXERCISE V CARDIOVASCULAR SOUNDS AND CASE STUDIES
1. Know the location for
auscultation for the aortic, pulmonary, bicuspid and tricuspid valves.
2. What happens to S2
when
you inhale deeply?
3. What type of murmurs
could best be heard during a deep inspiration?
4. What type of murmurs
are heard during diastole?
5. What type of murmurs
are heard during systole?
6. Review the Heart
Sounds
Case Studies: can you answer all of the questions? do you
understand?
LAB MANUAL: EXERCISE VI BLOOD PRESSURE, PULSE AND CASE STUDIES
1. Know the procedure for
taking blood pressure.
2. Be able to calculate
the pulse pressure and the mean arterial pressure.
3. What is the apical
pulse?
a pulse deficit?
4. Know the formulae on
the back page of the blood pressure case studies. Be prepared to
answer questions based on the concept or to do simple calculations
using
the formulae.
LAB MANUAL: EXERCISE VII BIOPAC EXERCISES
Baroreceptor Reflex
1.
Be able to draw and label a pulse pressure diagram.
How
is the pulse pressure used in physiology lab? What are the units
that are used for comparisons?
a. The pulse pressure is used in the lab to determine if there are
changes
in TPR.
If the amplitude of the curve increases then resistance decreases and if
the amplitude decreases resistance increases.
b. microvolts
2.
Be able to answer the questions in the lab manual related to the
baroreceptor
reflex.
1. What cardiovascular changes occur when the body changes from supine
to standing?
Hydrostatic pressure causes blood to move toward the feet and pool
there.
This
causes a decrease in venous return = decrease in cardiac output =
decrease
in
arterial BP
2. Where are baroreceptors located?
Aorta and carotid arteries
3. How does the baroreceptor reflex
counteract
the changes that result from the
positional change? The
drop
in pressure causes a decrease in input from the
receptors to the vasomotor center. A mass sympathetic response
occurs
which
increases HR., TPR, C.O. thus an increase in blood pressure
4. How does the 5 minute recovery data differ from the baseline supine
data?
Generally, the HR, TPR and BP will be slightly higher standing than in
the supine
postion
5. Were there any changes in heart rate and TPR from the beginning to
the
end of the
experimental data? At the beginning
of the recording we were seeing the end of
the baroreceptor response. As the blood pressure increased the
sympathetic
input decreased quickly to a level to sustain normal blood
pressure.
6. Why did the peripheral pulse
pressure
change when the position changed from
supine to standing? - ---- Since the
peripheral
pulse pressure is an indication of
flow, as the TPR increased the flow decreased and the amplitude of the
peripheral
pulse pressure decreased.
7. Why did some of the CV changes seen
after
standing persist after the five minute
recovery period? The changes
that
persist (higher HR and BP) allow normal CV
function and overcome the effect of hydrostatic pressure.
Cold Pressor Reflex
1. Read the information in the lab introduction.
2.
Answer the questions in the lab manual relating to the cold pressor
reflex.
1. By looking at your data, could you tell if
the water used in the lab was above or
below 15 degrees C?
If you saw a consistent increase in TPR the water was
above and if you saw a regular metabolic vasodilation then constriction
then
vasodilation etc. then the water was below 15 degrees C.
2. Why would vasodilation alternate with vasoconstriction when an
extremity
remains in very cold water? The
constriction is so severe that there is not
enough flow to maintain metabolism. There is periodic local flow
vasodilation
and then constriction then vasodilation, etc.
3. How fast did the cold pressor reflex act?From
looking at the recordings an
increase in TPR can be seen within two or three heartbeats.
4. Given the speed at which the cold pressor reflex acts, would you
consider
this an
important or minor reflex. This
is an important reflex used to control core body
temperature.
Copyright, Kay
Brashear
and James B. Parker, 1999